Genomic exploration of the targets of FOXL2 and ESR2 unveils their implication in cell migration, invasion, and adhesion
Author:
Affiliation:
1. Faculté des Sciences Université de Paris Paris France
2. Institut Jacques Monod Université de Paris, CNRS Paris France
3. Institut de Biologie F. Jacob Université Paris‐Saclay, Commissariat à l’Energie Atomique Fontenay aux Roses France
Funder
Agence Nationale de la Recherche
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202002444R
Reference45 articles.
1. The transcription factor FOXL2 mobilizes estrogen signaling to maintain the identity of ovarian granulosa cells
2. The murine winged-helix transcription factor Foxl2 is required for granulosa cell differentiation and ovary maintenance
3. Somatic Sex Reprogramming of Adult Ovaries to Testes by FOXL2 Ablation
4. The putative forkhead transcription factor FOXL2 is mutated in blepharophimosis/ptosis/epicanthus inversus syndrome
5. Towards a functional classification of pathogenic FOXL2 mutations using transactivation reporter systems
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