Mss51 deletion increases endurance and ameliorates histopathology in the mdx mouse model of Duchenne muscular dystrophy-Reference-Cited by-同舟云学术

Mss51 deletion increases endurance and ameliorates histopathology in the mdx mouse model of Duchenne muscular dystrophy

Published:2021-01-10 Issue:2 Volume:35 Page:
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Rovira Gonzalez Yazmin I.¹²^ORCID,Moyer Adam L.¹²,LeTexier Nicolas J.¹,Bratti August D.¹,Feng Siyuan¹,Peña Vanessa¹,Sun Congshan¹³,Pulcastro Hannah¹,Liu Ting⁴,Iyer Shama R.⁵,Lovering Richard M.⁵,O'Rourke Brian⁴,Wagner Kathryn R.¹³

Affiliation:

1. The Hugo W. Moser Research Institute Kennedy Krieger Institute Baltimore MD USA

2. Cellular and Molecular Medicine Graduate Program Johns Hopkins School of Medicine Baltimore MD USA

3. Departments of Neurology and Neuroscience Johns Hopkins School of Medicine Baltimore MD USA

4. Division of Cardiology Johns Hopkins University School of Medicine Baltimore MD USA

5. Department of Orthopaedics University of Maryland School of Medicine Baltimore MD USA

Funder

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202002106RR

Reference56 articles.

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2. Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals

3. Immunostaining of skeletal and cardiac muscle surface membrane with antibody against Duchenne muscular dystrophy peptide

4. Humanizing the mdx mouse model of DMD: the long and the short of it. npj;Yucel N;Regen Med,2018

5. Primary structure of dystrophin-associated glycoproteins linking dystrophin to the extracellular matrix

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