Inhibition of the PI 3‐kinase pathway disrupts the unfolded protein response and reduces sensitivity to ER stress‐dependent apoptosis-Reference-Cited by-同舟云学术

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Inhibition of the PI 3‐kinase pathway disrupts the unfolded protein response and reduces sensitivity to ER stress‐dependent apoptosis

Published:2020-08-03 Issue:9 Volume:34 Page:12521-12532
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Winnay Jonathon N.¹,Solheim Marie H.¹²,Sakaguchi Masaji¹³,Njølstad Pål R.²⁴,Kahn C. Ronald¹

Affiliation:

1. Joslin Diabetes Center Harvard Medical School Boston MA USA

2. Department of Clinical Science KG Jebsen Center for Diabetes Research University of Bergen Bergen Norway

3. Department of Metabolic Medicine Kumamoto University Kumamoto Japan

4. Department of Pediatrics Haukeland University Hospital Bergen Norway

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

FP7 Ideas: European Research Council

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.202000892R

Reference45 articles.

1. Signalling through Class I PI3Ks in mammalian cells

2. Five isoforms of the phosphatidylinositol 3-kinase regulatory subunit exhibit different associations with receptor tyrosine kinases and their tyrosine phosphorylations

3. p85α Gene Generates Three Isoforms of Regulatory Subunit for Phosphatidylinositol 3-Kinase (PI 3-Kinase), p50α, p55α, and p85α, with Different PI 3-Kinase Activity Elevating Responses to Insulin

4. The PI3K/AKT Pathway as a Target for Cancer Treatment

5. Idelalisib — A PI3Kδ Inhibitor for B-Cell Cancers

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