Pancreatic β‐cell Raf‐1 is required for glucose tolerance, insulin secretion, andinsulin 2transcription-Reference-Cited by-同舟云学术

Pancreatic β‐cell Raf‐1 is required for glucose tolerance, insulin secretion, andinsulin 2transcription

Published:2011-08-04 Issue:11 Volume:25 Page:3884-3895
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Alejandro Emilyn U.¹²,Lim Gareth E.¹²,Mehran Arya E.¹²,Hu Xiaoke¹²,Taghizadeh Farnaz¹²,Pelipeychenko Dmytro¹²,Baccarini Manuela³,Johnson James D.¹²

Affiliation:

1. Laboratory of Molecular Signalling in Diabetes, Diabetes Research Group, Department of Cellular and Physiological SciencesUniversity of British Columbia Vancouver BC Canada

2. Department of SurgeryUniversity of British Columbia Vancouver BC Canada

3. Max F. Perutz LaboratoriesUniversity of Vienna Vienna Austria

Funder

Juvenile Diabetes Research Foundation International

Canadian Diabetes Association

Canadian Institutes of Health Research

National Institutes of Health

University of British Columbia

Michael Smith Foundation for Health Research

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.10-180349

Reference52 articles.

1. Insulin Stimulates Primary β-Cell Proliferation via Raf-1 Kinase

2. Inhibition of Raf-1 Alters Multiple Downstream Pathways to Induce Pancreatic β-Cell Apoptosis

3. Acute Insulin Signaling in Pancreatic Beta-Cells Is Mediated by Multiple Raf-1 Dependent Pathways

4. EGF regulates survivin stability through the Raf‐1/ERK pathway in insulin‐secreting pancreatic beta‐cells;Wang H.;BMC Mol. Biol.,2010

5. Protein Farnesylation–Dependent Raf/Extracellular Signal–Related Kinase Signaling Links to Cytoskeletal Remodeling to Facilitate Glucose-Induced Insulin Secretion in Pancreatic β-Cells

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