Advanced glycation end products activate Smad signaling via TGF‐β‐dependent and ‐independent mechanisms: implications for diabetic renal and vascular disease-Reference-Cited by-同舟云学术

Advanced glycation end products activate Smad signaling via TGF‐β‐dependent and ‐independent mechanisms: implications for diabetic renal and vascular disease

Published:2003-04-22 Issue:1 Volume:18 Page:176-178
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Li Jin H.¹,Huang Xiao R.¹,Zhu Hong‐Jian²,Oldfield Matthew³,Cooper Mark³,Truong Luan D.¹,Johnson Richard J.¹,Lan Hui Y.¹

Affiliation:

1. Departments of Medicine‐Nephrology and PathologyBaylor College of Medicine Houston TX

2. Ludwig Institute for Cancer ResearchRoyal Melbourne Hospital Melbourne Australia

3. Department of Medicine, Austin and Repatriation Medical CenterThe University of Melbourne Melbourne Australia

Funder

National Health and Medical Research Council

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.02-1117fje

Reference43 articles.

1. RAGE: A New Target for the Prevention and Treatment of the Vascular and Inflammatory Complications of Diabetes

2. Advanced glycosylation end products in diabetic renal and vascular disease

3. An agent cleaving glucose-derived protein crosslinks in vitro and in vivo

4. Cellular receptors for advanced glycation end products. Implications for induction of oxidant stress and cellular dysfunction in the pathogenesis of vascular lesions.

5. Mechanisms of diabetic vasculopathy: an overview

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