Abnormal PTEN expression in portal hypertensive gastric mucosa: A key to impaired PI 3‐kinase/Akt activation and delayed injury healing?-Reference-Cited by-同舟云学术

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Abnormal PTEN expression in portal hypertensive gastric mucosa: A key to impaired PI 3‐kinase/Akt activation and delayed injury healing?

Published:2003-10-02 Issue:15 Volume:17 Page:2316-2318
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Tsugawa Kouji¹²,Jones Michael K.¹,Akahoshi Tomohiko¹²,Moon Woo Sung¹,Maewhara Yoshihiko²,Hashizume Makoto³,Sarfeh I. James¹,Tarnawski Andrzej S.¹

Affiliation:

1. Departments of Medicine and Surgery, Department of Veterans Affairs Medical Center, Long Beach, CaliforniaUniversity of CaliforniaIrvineCalifornia

2. Department of Surgery and Science, Graduate School of Medical ScienceKyushu UniversityFukuokaJapan

3. Department of Disaster and Emergency Medicine, Graduate School of Medical ScienceKyushu UniversityFukuokaJapan

Funder

U.S. Department of Veterans Affairs

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.02-1107fje

Reference48 articles.

1. The PTEN/MMAC1 tumor suppressor phosphatase functions as a negative regulator of the phosphoinositide 3-kinase/Akt pathway

2. New insights into tumor suppression: PTEN suppresses tumor formation by restraining the phosphoinositide 3-kinase/AKT pathway

3. The Tumor Suppressor, PTEN/MMAC1, Dephosphorylates the Lipid Second Messenger, Phosphatidylinositol 3,4,5-Trisphosphate

4. Protein Kinase B Is Expressed in Pancreatic β Cells and Activated upon Stimulation with Insulin-like Growth Factor I

5. Roles of phosphatidylinositol 3-kinase and mitogen- activated protein kinase pathways in stimulation of vascular smooth muscle cell migration and deoxyribonucleic acid synthesis by insulin-like growth factor-I;Imai Y.;Endocrinology,1999

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3. PTEN Inhibition in Human Disease Therapy;Molecules;2018-01-30

4. Downregulation of dickkopf-1 enhances the proliferation and osteogenic potential of fibroblasts isolated from ankylosing spondylitis patients via the Wnt/β-catenin signaling pathway in vitro;Connective Tissue Research;2016-03-16

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