Calcitonin induces connective tissue growth factor through ERK1/2 signaling in renal tubular cells
Author:
Affiliation:
1. Department of Pathology, Wakayama Medical University, Wakayama 641-0012, Japan.
2. Department of Rehabilitation, Osaka Kawasaki Rehabilitation University, Osaka 597-0104, Japan.
Publisher
Springer Science and Business Media LLC
Subject
Clinical Biochemistry,Molecular Biology,Molecular Medicine,Biochemistry
Reference36 articles.
1. Natriuretic Effect of Calcitonin in Man
2. In vitro evidence for differential involvement of CTGF, TGFβ, and PDGF‐BB in mesangial response to injury
3. The modular architecture of a new family of growth regulators related to connective tissue growth factor
4. Neither ERK nor JNK/SAPK MAP kinase subtypes are essential for histone H3/HMG-14 phosphorylation or c-fos and c-jun induction
5. Cell cycle-dependent and kinase-specific regulation of the apical Na/H exchanger and the Na,K-ATPase in the kidney cell line LLC-PK1 by calcitonin.
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