Naringenin Ameliorates Behavioral Dysfunction and Neurological Deficits in a d-Galactose-Induced Aging Mouse Model Through Activation of PI3K/Akt/Nrf2 Pathway-Reference-Cited by-同舟云学术

Naringenin Ameliorates Behavioral Dysfunction and Neurological Deficits in a d-Galactose-Induced Aging Mouse Model Through Activation of PI3K/Akt/Nrf2 Pathway

Published:2017-12 Issue:6 Volume:20 Page:462-472
ISSN:1549-1684
Container-title:Rejuvenation Research
language:en
Short-container-title:Rejuvenation Research

Author:

Zhang Yan¹,Liu Bin¹,Chen Xi²³,Zhang Ning¹,Li Guang³,Zhang Li-Hong¹,Tan Li-Yan⁴

Affiliation:

1. Jiamusi College, Heilongjiang University of Chinese Medicine, Jiamusi, China.

2. Institute of Medicinal Plant Development, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing, China.

3. Yunnan Branch, Institute of Medicinal Plant Development, Chinese Academy of Medical Sciences and Peking Union Medical College, Jing Hong, China.

4. Department of Endocrinology, The First Affiliated Hospital of Jiamusi University, Jiamusi, China.

Publisher

Mary Ann Liebert Inc

Subject

Geriatrics and Gerontology,Ageing

Link

https://www.liebertpub.com/doi/pdf/10.1089/rej.2017.1960

Reference41 articles.

1. Aging: A Theory Based on Free Radical and Radiation Chemistry

2. Oxidants, oxidative stress and the biology of ageing

3. Oxidative damage in Alzheimer's disease: the metabolic dimension

4. Redox imbalance influence in the myocardial Akt activation in aged rats treated with DHEA

5. Role of NRF2 in Protection Against Hyperoxic Lung Injury in Mice

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