Redox Regulation of Pro-IL-1β Processing May Contribute to the Increased Severity of Serum-Induced Arthritis in NOX2-Deficient Mice-Reference-Cited by-同舟云学术

Redox Regulation of Pro-IL-1β Processing May Contribute to the Increased Severity of Serum-Induced Arthritis in NOX2-Deficient Mice

Published:2015-10-20 Issue:12 Volume:23 Page:973-984
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Huang Ya-Fang¹,Lo Pei-Chi¹,Yen Chia-Liang²,Nigrovic Peter Andrija³⁴,Chao Wen-Chen¹⁵,Wang Wei-Zhi¹,Hsu George Chengkang¹,Tsai Yau-Sheng¹,Shieh Chi-Chang¹⁶

Affiliation:

1. Institute of Clinical Medicine, National Cheng Kung University College of Medicine, Tainan, Taiwan.

2. Institute of Basic Medical Science, National Cheng Kung University College of Medicine, Tainan, Taiwan.

3. Division of Rheumatology, Immunology, and Allergy, Brigham and Women's Hospital, Boston, Massachusetts.

4. Division of Immunology, Boston Children's Hospital, Boston, Massachusetts.

5. Department of Internal Medicine, Taichung Veteran General Hospital, Chiayi Branch, Chiayi, Taiwan.

6. Department of Pediatrics, National Cheng Kung University Hospital, Tainan, Taiwan.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2014.6136

Reference62 articles.

1. Reactive oxygen species and superoxide dismutases: Role in joint diseases

2. NALP3 Forms an IL-1β-Processing Inflammasome with Increased Activity in Muckle-Wells Autoinflammatory Disorder

3. The Neutrophil NADPH Oxidase

4. Subtype-specific peripheral blood gene expression profiles in recent-onset juvenile idiopathic arthritis

5. Human CD4+CD3−Innate-Like T Cells Provide a Source of TNF and Lymphotoxin-αβ and Are Elevated in Rheumatoid Arthritis

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