NOX4 NADPH Oxidase-Dependent Mitochondrial Oxidative Stress in Aging-Associated Cardiovascular Disease-Reference-Cited by-同舟云学术

NOX4 NADPH Oxidase-Dependent Mitochondrial Oxidative Stress in Aging-Associated Cardiovascular Disease

Published:2015-12-20 Issue:18 Volume:23 Page:1389-1409
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Vendrov Aleksandr E.¹,Vendrov Kimberly C.²,Smith Alberto³,Yuan Jinling¹,Sumida Arihiro¹,Robidoux Jacques⁴,Runge Marschall S.¹,Madamanchi Nageswara R.¹

Affiliation:

1. Department of Medicine, McAllister Heart Institute, University of North Carolina, Chapel Hill, North Carolina.

2. Division of Pharmacotherapy and Experimental Therapeutics, Eshelman School of Pharmacy, University of North Carolina, Chapel Hill, North Carolina.

3. Cardiovascular Division, Academic Department of Surgery, National Institute for Health Research Biomedical Research Center at Guy's and St Thomas' National Health Service Foundation Trust, King's College London British Heart Foundation Centre of Excellence, London, United Kingdom.

4. Department of Pharmacology and Toxicology, The East Carolina Diabetes and Obesity Institute, East Carolina University, Greenville, North Carolina.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2014.6221

Reference67 articles.

1. Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

2. The LDLR deficient mouse as a model for aortic calcification and quantification by micro-computed tomography

3. A far-upstream AP-1/Smad binding box regulates human NOX4 promoter activation by transforming growth factor-β

4. Mitochondrial Integrity and Function in Atherogenesis

5. p47phox is required for atherosclerotic lesion progression in ApoE–/– mice

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