Iron in Neurodegenerative Disorders of Protein Misfolding: A Case of Prion Disorders and Parkinson's Disease
Author:
Affiliation:
1. Department of Pathology, Case Western Reserve University, Cleveland, Ohio.
2. School of Medicine, Case Western Reserve University, Cleveland, Ohio.
3. School of Arts and Sciences, Case Western Reserve University, Cleveland, Ohio.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry
Link
http://www.liebertpub.com/doi/pdf/10.1089/ars.2014.5874
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1. Autophagy induction by trehalose counter-acts cellular prion-infection
2. The dopamine metabolite aminochrome inhibits mitochondrial complex I and modifies the expression of iron transporters DMT1 and FPN1
3. Prions: Protein Aggregation and Infectious Diseases
4. Prion propagation, toxicity and degradation
5. Protein aggregation diseases: pathogenicity and therapeutic perspectives
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