Extracellular Vesicles Attenuate Nitrofen-Mediated Human Pulmonary Artery Endothelial Dysfunction: Implications for Congenital Diaphragmatic Hernia-Reference-Cited by-同舟云学术

Extracellular Vesicles Attenuate Nitrofen-Mediated Human Pulmonary Artery Endothelial Dysfunction: Implications for Congenital Diaphragmatic Hernia

Published:2020-08-01 Issue:15 Volume:29 Page:967-980
ISSN:1547-3287
Container-title:Stem Cells and Development
language:en
Short-container-title:Stem Cells and Development

Author:

Zhaorigetu Siqin¹,Bair Henry¹²,Jin Di¹,Gupta Vikas S.¹,Pandit Lavannya M.³,Bryan Robert M.³,Lally Kevin P.¹,Olson Scott D.¹,Cox Charles S.¹,Harting Matthew T.¹

Affiliation:

1. Department of Pediatric Surgery, McGovern Medical School at the University of Texas Health Science Center (UTHealth) and Children's Memorial Hermann Hospital, Houston, Texas, USA.

2. Stanford University School of Medicine, Stanford, California, USA.

3. Baylor College of Medicine and Michael E. DeBakey Veterans Affairs Medical Center, Houston, Texas, USA.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Developmental Biology,Hematology

Link

https://www.liebertpub.com/doi/pdf/10.1089/scd.2020.0063

Reference66 articles.

1. Recent advances in the management of congenital diaphragmatic hernia

2. Enhanced Pulmonary Vascular and Alveolar Development via Prenatal Administration of a Slow-Release Synthetic Prostacyclin Agonist in Rat Fetal Lung Hypoplasia

3. Perturbations in Endothelial Dysfunction-Associated Pathways in the Nitrofen-Induced Congenital Diaphragmatic Hernia Model