p62/SQSTM1 and Selective Autophagy in Cardiometabolic Diseases-Reference-Cited by-同舟云学术

p62/SQSTM1 and Selective Autophagy in Cardiometabolic Diseases

Published:2019-08-20 Issue:6 Volume:31 Page:458-471
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Jeong Se-Jin¹,Zhang Xiangyu¹,Rodriguez-Velez Astrid¹,Evans Trent D.¹,Razani Babak¹²³

Affiliation:

1. Cardiovascular Division, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri.

2. Department of Pathology and Immunology, Washington University School of Medicine, St. Louis, Missouri.

3. John Cochran VA Medical Center, St. Louis, Missouri.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2018.7649

Reference113 articles.

1. The eIF2α/ATF4 pathway is essential for stress-induced autophagy gene expression

2. TRIM16 Acts as an E3 Ubiquitin Ligase and Can Heterodimerize with Other TRIM Family Members

3. A Proximity Labeling Strategy Provides Insights into the Composition and Dynamics of Lipid Droplet Proteomes

4. Enhanced autophagy ameliorates cardiac proteinopathy

5. The mitochondrial deubiquitinase USP30 opposes parkin-mediated mitophagy

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