Follistatin Protects Against Glomerular Mesangial Cell Apoptosis and Oxidative Stress to Ameliorate Chronic Kidney Disease-Reference-Cited by-同舟云学术

Follistatin Protects Against Glomerular Mesangial Cell Apoptosis and Oxidative Stress to Ameliorate Chronic Kidney Disease

Published:2019-09-10 Issue:8 Volume:31 Page:551-571
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Mehta Neel¹,Gava Agata L.²,Zhang Dan¹,Gao Bo¹,Krepinsky Joan C.¹

Affiliation:

1. Division of Nephrology, Department of Medicine, McMaster University, Hamilton, Canada.

2. Physiological Sciences Graduate Program, Health Sciences Centre, Federal University of Espirito Santo, Vitoria, Brazil.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2018.7684

Reference75 articles.

1. Ligand trap for the activin type IIA receptor protects against vascular disease and renal fibrosis in mice with chronic kidney disease

2. Pyocyanin Production byPseudomonas aeruginosaInduces Neutrophil Apoptosis and Impairs Neutrophil-Mediated Host Defenses In Vivo

3. Attenuation of Bleomycin-induced Pulmonary Fibrosis by Follistatin

4. A new model of diabetic nephropathy in C57BL/6 mice challenged with advanced oxidation protein products

5. NAD(P)H Oxidase Mediates TGF-β1–Induced Activation of Kidney Myofibroblasts

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