Important Role of Sarcoplasmic Reticulum Ca2+ Release via Ryanodine Receptor-2 Channel in Hypoxia-Induced Rieske Iron–Sulfur Protein-Mediated Mitochondrial Reactive Oxygen Species Generation in Pulmonary Artery Smooth Muscle Cells-Reference-Cited by-同舟云学术

Important Role of Sarcoplasmic Reticulum Ca2+ Release via Ryanodine Receptor-2 Channel in Hypoxia-Induced Rieske Iron–Sulfur Protein-Mediated Mitochondrial Reactive Oxygen Species Generation in Pulmonary Artery Smooth Muscle Cells

Published:2020-03-01 Issue:7 Volume:32 Page:447-462
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Yang Zhao¹²,Song Tengyao¹,Truong Lillian¹,Reyes-García Jorge¹,Wang Lan¹,Zheng Yun-Min¹,Wang Yong-Xiao¹

Affiliation:

1. Department of Molecular and Cellular Physiology, Albany Medical College, Albany, New York.

2. Department of Respiratory Medicine, Suzhou Science & Technology Town Hospital, Suzhou, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2018.7652

Reference53 articles.

1. Genetically encoded fluorescent indicator for intracellular hydrogen peroxide

2. Enhancement of hydrogen peroxide formation by protophores and ionophores in antimycin-supplemented mitochondria

3. Reactive Oxygen Species Generated at Mitochondrial Complex III Stabilize Hypoxia-inducible Factor-1α during Hypoxia

4. Directly interact with Keap1 and LPS is involved in the anti-inflammatory mechanisms of (-)-epicatechin-3-gallate in LPS-induced macrophages and endotoxemia

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