Aluminum Modulates Effects ofβAmyloid1–42on Neuronal Calcium Homeostasis and Mitochondria Functioning and Is Altered in a Triple Transgenic Mouse Model of Alzheimer's Disease-Reference-Cited by-同舟云学术

Aluminum Modulates Effects ofβAmyloid1–42on Neuronal Calcium Homeostasis and Mitochondria Functioning and Is Altered in a Triple Transgenic Mouse Model of Alzheimer's Disease

Published:2008-10 Issue:5 Volume:11 Page:861-871
ISSN:1549-1684
Container-title:Rejuvenation Research
language:en
Short-container-title:Rejuvenation Research

Author:

Drago Denise¹,Cavaliere Alessandra¹,Mascetra Nicola²,Ciavardelli Domenico³,Di Ilio Carmine³,Zatta Paolo¹,Sensi Stefano L.²⁴

Affiliation:

1. CNR-Institute for Biomedical Technologies, Padua “Metalloproteins” Unit, Department of Biology, University of Padua, Padua, Italy.

2. Department of Basic and Applied Medical Science, Molecular Neurology Unit, CeSI-Center for Excellence on Aging, University ‘G. d'Annunzio’, Chieti, Italy.

3. Department of Biochemistry, Biochemistry Unit, CeSI-Center for Excellence on Aging, University ‘G. d'Annunzio’, Chieti, Italy.

4. Department of Neurology, University of California–Irvine, Irvine, California.

Publisher

Mary Ann Liebert Inc

Subject

Geriatrics and Gerontology,Ageing

Link

https://www.liebertpub.com/doi/pdf/10.1089/rej.2008.0761

Reference69 articles.

1. The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics

2. Oligomeric and Fibrillar Species of Amyloid-β Peptides Differentially Affect Neuronal Viability

3. The synaptic Aβ hypothesis of Alzheimer disease