High-Affinity Interaction between HIV-1 Vpr and Specific Sequences That Span the C/EBP and Adjacent NF-κB Sites within the HIV-1 LTR Correlate with HIV-1-Associated Dementia-Reference-Cited by-同舟云学术

High-Affinity Interaction between HIV-1 Vpr and Specific Sequences That Span the C/EBP and Adjacent NF-κB Sites within the HIV-1 LTR Correlate with HIV-1-Associated Dementia

Published:2004-04 Issue:4 Volume:23 Page:261-269
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Burdo Tricia H.¹,Nonnemacher Michael¹,Irish Bryan P.²,Choi Catherine H.²,Krebs Fred C.²,Gartner Suzanne³,Wigdahl Brian²

Affiliation:

1. Department of Microbiology and Immunology, The Pennsylvania State University, College of Medicine, Hershey, Pennsylvania

2. Department of Microbiology and Immunology, and Institute for Molecular Medicine and Infectious Disease, Drexel University College of Medicine, Philadelphia, Pennsylvania

3. Department of Neurology, The Johns Hopkins University School of Medicine, Baltimore, Maryland

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/104454904773819842

Reference26 articles.

1. Brain-derived HIV-1 tat sequences from AIDS patients with dementia show increased molecular heterogeneity

2. Vpr Is Required for Efficient Replication of Human Immunodeficiency Virus Type-1 in Mononuclear Phagocytes

3. HIV-1 Vpr Enhances Viral Burden by Facilitating Infection of Tissue Macrophages but Not Nondividing CD4+ T Cells

4. HIV-1 Regulatory/Accessory Genes: Keys to Unraveling Viral and Host Cell Biology

5. HIV transcriptional activation by the accessory protein, VPR, is mediated by the p300 co-activator

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