Phenotypic Differentiation without Permanent Cell-Cycle Arrest by Skeletal Myocytes with Deregulated E2F-1
Author:
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/104454999315367
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1. Deficiency of retinoblastoma protein leads to inappropriate S-phase entry, activation of E2F-responsive genes, and apoptosis.
2. Myogenin expression, cell cycle withdrawal, and phenotypic differentiation are temporally separable events that precede cell fusion upon myogenesis.
3. The retinoblastoma protein copurifies with E2F-I, an E1A-regulated inhibitor of the transcription factor E2F
4. E2F-4, a new member of the E2F gene family, has oncogenic activity and associates with p107 in vivo.
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