Underlying Mechanisms and Related Diseases Behind the Complex Regulatory Role of NOD-Like Receptor X1
Author:
Affiliation:
1. Department of Spine Surgery, Zhongda Hospital, School of Medicine, Southeast University, Nanjing, China.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
https://www.liebertpub.com/doi/pdf/10.1089/dna.2022.0051
Reference104 articles.
1. Enhancement of Reactive Oxygen Species Production and Chlamydial Infection by the Mitochondrial Nod-like Family Member NLRX1
2. NLRX1 Negatively Regulates Group A Streptococcus Invasion and Autophagy Induction by Interacting With the Beclin 1–UVRAG Complex
3. Pathogen Recognition and Innate Immunity
4. NLRX1 Protein Attenuates Inflammatory Responses to Infection by Interfering with the RIG-I-MAVS and TRAF6-NF-κB Signaling Pathways
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2. NLRX1 Inhibits LPS-Induced Microglial Death via Inducing p62-Dependent HO-1 Expression, Inhibiting MLKL and Activating PARP-1;Antioxidants;2024-04-17
3. A zebrafish NLRX1 isoform downregulates fish IFN responses by targeting the adaptor STING;Journal of Virology;2024-02-20
4. Cellular and Molecular Mechanisms Involving Synbiotics in Various Disease State;Synbiotics in Human Health: Biology to Drug Delivery;2024
5. NLRX1 increases human retinal pigment epithelial autophagy and reduces H2O2-induced oxidative stress and inflammation by suppressing FUNDC1 phosphorylation and NLRP3 activation;Allergologia et Immunopathologia;2023-01-01
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