Adenosine A2A Receptor Antagonist Sch58261 Improves the Cognitive Function in Alzheimer’s Disease Model Mice Through Activation of Nrf2 via an Autophagy-Dependent Pathway-Reference-Cited by-同舟云学术

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Adenosine A2A Receptor Antagonist Sch58261 Improves the Cognitive Function in Alzheimer’s Disease Model Mice Through Activation of Nrf2 via an Autophagy-Dependent Pathway

Published:2024-07-08 Issue: Volume: Page:
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Sun Yi¹,Liu Chao¹²,He Ling¹^ORCID

Affiliation:

1. Department of Pharmacology, School of Pharmacy, China Pharmaceutical University, Nanjing, China.

2. Jiangsu Key Laboratory of Brain Disease Bioinformation, Research Center for Biochemistry and Molecular Biology, Xuzhou Medical University, Xuzhou, China.

Publisher

Mary Ann Liebert Inc

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2023.0455

Reference58 articles.

1. 2022 Alzheimer's disease facts and figures

2. RESEARCH ARTICLE: Up-regulation of Adenosine Receptors in the Frontal Cortex in Alzheimer's Disease

3. Pharmacology of Adenosine Receptors: The State of the Art

4. Synergy between amyloid-β and tau in Alzheimer’s disease

5. Adenosine A2A Receptor Blockade Prevents Synaptotoxicity and Memory Dysfunction Caused by -Amyloid Peptides via p38 Mitogen-Activated Protein Kinase Pathway

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