Thyroid Antigens, Not Central Tolerance, Control Responses to Immunization in BALB/c Versus C57BL/6 Mice
Author:
Affiliation:
1. Autoimmune Disease Unit, Cedars-Sinai Medical Center and UCLA Medical School, Los Angeles, California.
Publisher
Mary Ann Liebert Inc
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Link
https://www.liebertpub.com/doi/pdf/10.1089/thy.2008.0420
Reference43 articles.
1. Genetic immunization of outbred mice with thyrotropin receptor cDNA provides a model of Graves’ disease
2. CpG motifs in bacterial DNA trigger direct B-cell activation
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1. Nanoparticles Bearing TSH Receptor Protein and a Tolerogenic Molecule Do Not Induce Immune Tolerance but Exacerbate Thyroid Autoimmunity in hTSHR/NOD.H2h4 Mice;The Journal of Immunology;2019-04-03
2. Thyroid Autoantibodies Display both “Original Antigenic Sin” and Epitope Spreading;Frontiers in Immunology;2017-12-20
3. Aberrant Iodine Autoregulation Induces Hypothyroidism in a Mouse Strain in the Absence of Thyroid Autoimmunity;Journal of the Endocrine Society;2017-11-29
4. Critical Differences between Induced and Spontaneous Mouse Models of Graves’ Disease with Implications for Antigen-Specific Immunotherapy in Humans;The Journal of Immunology;2016-11-09
5. Thyrotropin Receptor Epitope and Human Leukocyte Antigen in Graves’ Disease;Frontiers in Endocrinology;2016-08-23
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