Demethylation of GFRA4 Promotes Cell Proliferation and Invasion in Hirschsprung Disease
Author:
Affiliation:
1. Department of Pediatric Surgery, Shandong Provincial Hospital Affiliated to Shandong University, Jinan, China.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/dna.2017.3928
Reference24 articles.
1. Genetic Background of Hirschsprung Disease: A Bridge Between Basic Science and Clinical Application
2. The GDNF family ligands and receptors — implications for neural development
3. Artemin, a Novel Member of the GDNF Ligand Family, Supports Peripheral and Central Neurons and Signals through the GFRα3–RET Receptor Complex
4. Investigation of germline GFRA4 mutations and evaluation of the involvement of GFRA1, GFRA2, GFRA3, and GFRA4 sequence variants in Hirschsprung disease
5. Neurturin responsiveness requires a GPI-linked receptor and the Ret receptor tyrosine kinase
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