Lack of the Thyroid Hormone Transporter Mct8 in Osteoblast and Osteoclast Progenitors Increases Trabecular Bone in Male Mice-Reference-Cited by-同舟云学术

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Lack of the Thyroid Hormone Transporter Mct8 in Osteoblast and Osteoclast Progenitors Increases Trabecular Bone in Male Mice

Published:2020-02-01 Issue:2 Volume:30 Page:329-342
ISSN:1050-7256
Container-title:Thyroid
language:en
Short-container-title:Thyroid

Author:

Lademann Franziska¹²,Tsourdi Elena¹²,Rijntjes Eddy³,Köhrle Josef³,Hofbauer Lorenz C.¹²,Heuer Heike⁴,Rauner Martina¹²

Affiliation:

1. Department of Medicine III, Universitätsklinikum Dresden, Dresden, Germany.

2. Center for Healthy Aging, Universitätsklinikum Dresden, Dresden, Germany.

3. Institut für Experimentelle Endokrinologie, Charité-Universitätsmedizin Berlin, Berlin, Germany.

4. Klinik für Endokrinologie, Universitätsklinikum Essen, Universität Duisburg-Essen, Essen, Germany.

Publisher

Mary Ann Liebert Inc

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Link

https://www.liebertpub.com/doi/pdf/10.1089/thy.2019.0271

Reference62 articles.

1. Bone remodelling at a glance

2. Cellular and Molecular Mechanisms of Bone Remodeling

3. Role of Thyroid Hormones in Skeletal Development and Bone Maintenance

4. Thyroid hormone metabolism in skeletal development and adult bone maintenance

5. Thyroid disorders---an update

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4. Reduced Bone Mass and Increased Osteocyte Tartrate‐Resistant Acid Phosphatase ( TRAP) Activity, But Not Low Mineralized Matrix Around Osteocyte Lacunae, Are Restored After Recovery From Exogenous Hyperthyroidism in Male Mice;Journal of Bone and Mineral Research;2022-11-22

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