Retracted: Tumor Protein D52-Like 2 Contributes to Proliferation of Breast Cancer Cells-Reference-Cited by-同舟云学术

Retracted: Tumor Protein D52-Like 2 Contributes to Proliferation of Breast Cancer Cells

Published:2015-02 Issue:1 Volume:30 Page:1-7
ISSN:1084-9785
Container-title:Cancer Biotherapy and Radiopharmaceuticals
language:en
Short-container-title:Cancer Biotherapy and Radiopharmaceuticals

Author:

Yang Mei¹,Wang Xueyao²,Jia Jiaoyuan³,Gao Hongwen⁴,Chen Peng⁵,Sha Xianliang⁴,Wu Shan¹

Affiliation:

1. The Key Laboratory of Pathobiology, Ministry of Education, Department of Pathology, College of Basic Medical Sciences, Norman Bethune Health Science Center of Jilin University, Changchun, China.

2. Norman Bethune Health Science Center of Jilin University, Changchun, China.

3. Department of Oncology and Hematology, The Second Hospital of Jilin University, Changchun, China.

4. Department of Pathology, The Second Hospital of Jilin University, Changchun, China.

5. Department of Pediatrics, The Second Hospital of Jilin University, Changchun, China.

Publisher

Mary Ann Liebert Inc

Subject

Cancer Research,Pharmacology,Radiology, Nuclear Medicine and imaging,Oncology,General Medicine,Cancer Research,Pharmacology,Radiology, Nuclear Medicine and imaging,Oncology,General Medicine

Link

https://www.liebertpub.com/doi/pdf/10.1089/cbr.2014.1723

Reference24 articles.

1. Comprehensive copy number profiles of breast cancer cell model genomes

2. Akt Blocks Breast Cancer Cell Motility and Invasion through the Transcription Factor NFAT

3. Mechanisms of Disease: understanding resistance to HER2-targeted therapy in human breast cancer

4. Identification of homo- and heteromeric interactions between members of the breast carcinoma-associated D52 protein family using the yeast two-hybrid system

5. D53 (TPD52L1) is a cell cycle-regulated protein maximally expressed at the G2-M transition in breast cancer cells

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