Intracerebroventricular High Doses of 3,3′,5-Triiodothyroacetic Acid at Juvenile Stages Improve Peripheral Hyperthyroidism and Mediate Thyromimetic Effects in Limited Brain Regions in a Mouse Model of Monocarboxylate Transporter 8 Deficiency-Reference-Cited by-同舟云学术

Intracerebroventricular High Doses of 3,3′,5-Triiodothyroacetic Acid at Juvenile Stages Improve Peripheral Hyperthyroidism and Mediate Thyromimetic Effects in Limited Brain Regions in a Mouse Model of Monocarboxylate Transporter 8 Deficiency

Published:2023-04-01 Issue:4 Volume:33 Page:501-510
ISSN:1050-7256
Container-title:Thyroid
language:en
Short-container-title:Thyroid

Author:

Grijota-Martínez Carmen¹²,Montero-Pedrazuela Ana¹,Hidalgo-Álvarez Jorge¹,Bárez-López Soledad¹^ORCID,Guadaño-Ferraz Ana¹^ORCID

Affiliation:

1. Department of Endocrine and Nervous System Pathophysiology, Instituto de Investigaciones Biomédicas “Alberto Sols,” Consejo Superior de Investigaciones Científicas (CSIC)-Universidad Autónoma de Madrid (UAM), Madrid, Spain.

2. Departamento de Biología Celular, Facultad de Ciencias Biológicas, Universidad Complutense de Madrid, Madrid, Spain.

Publisher

Mary Ann Liebert Inc

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Link

https://www.liebertpub.com/doi/pdf/10.1089/thy.2022.0562

Reference43 articles.

1. A Novel Syndrome Combining Thyroid and Neurological Abnormalities Is Associated with Mutations in a Monocarboxylate Transporter Gene

2. Association between mutations in a thyroid hormone transporter and severe X-linked psychomotor retardation

3. Monocarboxylate Transporter 8 Deficiency: Delayed or Permanent Hypomyelination?

4. Movement disorders in MCT8 deficiency/Allan-Herndon-Dudley Syndrome

5. Identification of Monocarboxylate Transporter 8 as a Specific Thyroid Hormone Transporter

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1. Unmet patient needs in monocarboxylate transporter 8 (MCT8) deficiency: a review;Frontiers in Pediatrics;2024-07-22

2. In a zebrafish biomedical model of human Allan-Herndon-Dudley syndrome impaired MTH signaling leads to decreased neural cell diversity;Frontiers in Endocrinology;2023-05-04