Friedreich Ataxia: Molecular Mechanisms, Redox Considerations, and Therapeutic Opportunities-Reference-Cited by-同舟云学术

Friedreich Ataxia: Molecular Mechanisms, Redox Considerations, and Therapeutic Opportunities

Published:2010-09 Issue:5 Volume:13 Page:651-690
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Santos Renata¹,Lefevre Sophie¹²,Sliwa Dominika¹,Seguin Alexandra¹,Camadro Jean-Michel¹,Lesuisse Emmanuel¹

Affiliation:

1. Mitochondria, Metals and Oxidative Stress Laboratory, Institut Jacques Monod (UMR 7592 CNRS–University Paris-Diderot), Paris, France.

2. University Pierre et Marie Curie, Paris, France.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.3015

Reference336 articles.

1. Friedreich's ataxia. A clinical review with neurophysiological and echocardiographic findings.

2. Recombinant Human Erythropoietin Increases Frataxin Protein Expression Without Increasing mRNA Expression

3. Extra-mitochondrial localisation of frataxin and its association with IscU1 during enterocyte-like differentiation of the human colon adenocarcinoma cell line Caco-2

4. Iron-Dependent Self-Assembly of Recombinant Yeast Frataxin: Implications for Friedreich Ataxia

5. Bacterial frataxin CyaY is the gatekeeper of iron-sulfur cluster formation catalyzed by IscS

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