Glutaredoxin Regulates Apoptosis in Cardiomyocytes via NFκB Targets Bcl-2 and Bcl-xL: Implications for Cardiac Aging-Reference-Cited by-同舟云学术

Glutaredoxin Regulates Apoptosis in Cardiomyocytes via NFκB Targets Bcl-2 and Bcl-xL: Implications for Cardiac Aging

Published:2010-06-15 Issue:12 Volume:12 Page:1339-1353
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Gallogly Molly M.¹,Shelton Melissa D.¹,Qanungo Suparna¹,Pai Harish V.¹,Starke David W.¹,Hoppel Charles L.¹,Lesnefsky Edward J.²³,Mieyal John J.¹²

Affiliation:

1. Department of Pharmacology, Case Western Reserve University, School of Medicine, Cleveland, Ohio.

2. Department of Internal Medicine, Division of Cardiology, Virginia Commonwealth University, and Medical Service, McGuire Veterans Affairs Medical Center, Richmond, Virginia.

3. Department of Medicine, Division of Cardiology, Case Western Reserve University and Medical Service, Veterans Affairs Medical Center, Cleveland, Ohio.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2791

Reference57 articles.

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2. Effect of NF- κ B Inhibition on TNF- α -induced Apoptosis and Downstream Pathways in Cardiomyocytes

3. H9c2 cardiac myoblasts undergo apoptosis in a model of ischemia consisting of serum deprivation and hypoxia: inhibition by PMA

4. Cardiac functional improvement by a human Bcl-2 transgene in a mouse model of ischemia/reperfusion injury

5. BMS-345541 Is a Highly Selective Inhibitor of IκB Kinase That Binds at an Allosteric Site of the Enzyme and Blocks NF-κB-dependent Transcription in Mice

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