Evidence Supporting a Role for Endoplasmic Reticulum Stress in the Development of Atherosclerosis in a Hyperglycaemic Mouse Model-Reference-Cited by-同舟云学术

Evidence Supporting a Role for Endoplasmic Reticulum Stress in the Development of Atherosclerosis in a Hyperglycaemic Mouse Model

Published:2009-09 Issue:9 Volume:11 Page:2289-2298
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Khan Mohammad I.¹²,Pichna Branislav A.³²,Shi Yuanyuan¹,Bowes Anna J.³,Werstuck Geoff H.¹³⁴

Affiliation:

1. Henderson Research Centre, McMaster University, Hamilton, Ontario, Canada.

2. These authors contributed equally to this study.

3. Department of Biochemistry and Biomedical Sciences, McMaster University, Hamilton, Ontario, Canada.

4. Department of Medicine, McMaster University, Hamilton, Ontario, Canada.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2569

Reference42 articles.

1. Valproate Attenuates Accelerated Atherosclerosis in Hyperglycemic ApoE-Deficient Mice

2. EIF2AK3, encoding translation initiation factor 2-α kinase 3, is mutated in patients with Wolcott-Rallison syndrome

3. Autoimmune Diabetes Induced by the β-cell Toxin STZ: Immunity to the 60-kDa Heat Shock Protein and to Insulin

4. ER quality control: towards an understanding at the molecular level

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