Macrophage Apoptosis in Atherosclerosis: Consequences on Plaque Progression and the Role of Endoplasmic Reticulum Stress-Reference-Cited by-同舟云学术

Macrophage Apoptosis in Atherosclerosis: Consequences on Plaque Progression and the Role of Endoplasmic Reticulum Stress

Published:2009-09 Issue:9 Volume:11 Page:2333-2339
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Tabas Ira¹

Affiliation:

1. Departments of Medicine, Pathology & Cell Biology, and Physiology & Cellular Biophysics, Columbia University, New York, New York.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2469

Reference49 articles.

1. A role for the apoptosis inhibitory factor AIM/Spα/Api6 in atherosclerosis development

2. Evidence that the death of macrophage foam cells contributes to the lipid core of atheroma

3. The cholesteryl ester cycle in macrophage foam cells. Continual hydrolysis and re-esterification of cytoplasmic cholesteryl esters.

4. Morphologic Findings of Coronary Atherosclerotic Plaques in Diabetics

5. Pivotal Advance: Macrophages become resistant to cholesterol-induced death after phagocytosis of apoptotic cells

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