Lithium Ameliorates Neurodegeneration, Suppresses Neuroinflammation, and Improves Behavioral Performance in a Mouse Model of Traumatic Brain Injury-Reference-Cited by-同舟云学术

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Lithium Ameliorates Neurodegeneration, Suppresses Neuroinflammation, and Improves Behavioral Performance in a Mouse Model of Traumatic Brain Injury

Published:2012-01-20 Issue:2 Volume:29 Page:362-374
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Yu Fengshan¹²³,Wang Zhifei¹,Tchantchou Flaubert³,Chiu Chi-Tso¹,Zhang Yumin²³,Chuang De-Maw¹²

Affiliation:

1. Section on Molecular Neurobiology, National Institute of Mental Health, National Institutes of Health, Bethesda, Maryland.

2. Center for Neuroscience and Regenerative Medicine, Uniformed Services University of the Health Sciences, Bethesda, Maryland.

3. Department of Anatomy, Physiology and Genetics, Uniformed Services University of the Health Sciences, Bethesda, Maryland.

Publisher

Mary Ann Liebert Inc

Subject

Clinical Neurology

Link

http://www.liebertpub.com/doi/pdf/10.1089/neu.2011.1942

Reference59 articles.

1. Selective Loss of Brain-Derived Neurotrophic Factor in the Dentate Gyrus Attenuates Antidepressant Efficacy

2. Genetic disruption of cyclooxygenase-2 does not improve histological or behavioral outcome after traumatic brain injury in mice

3. Emerging roles of PGE2 receptors in models of neurological disease

4. Lithium antagonizes dopamine-dependent behaviors mediated by an AKT/glycogen synthase kinase 3 signaling cascade

5. Innate and adaptive immune responses regulated by glycogen synthase kinase-3 (GSK3)

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