Post-Injury Administration of the Mitochondrial Permeability Transition Pore Inhibitor, NIM811, Is Neuroprotective and Improves Cognition after Traumatic Brain Injury in Rats-Reference-Cited by-同舟云学术

Post-Injury Administration of the Mitochondrial Permeability Transition Pore Inhibitor, NIM811, Is Neuroprotective and Improves Cognition after Traumatic Brain Injury in Rats

Published:2011-09 Issue:9 Volume:28 Page:1845-1853
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Readnower Ryan D.¹²,Pandya Jignesh D.¹,McEwen Melanie L.¹²³,Pauly James R.¹⁴,Springer Joseph E.¹²³,Sullivan Patrick G.¹²

Affiliation:

1. Spinal Cord & Brain Injury Research Center, University of Kentucky, Lexington, Kentucky.

2. Department of Anatomy and Neurobiology, University of Kentucky, Lexington, Kentucky.

3. Department of Physical Medicine and Rehabilitation, University of Kentucky, Lexington, Kentucky.

4. Department of Pharmaceutical Sciences, University of Kentucky, Lexington, Kentucky.

Publisher

Mary Ann Liebert Inc

Subject

Clinical Neurology

Link

http://www.liebertpub.com/doi/pdf/10.1089/neu.2011.1755

Reference61 articles.

1. Cyclosporin A Improves Brain Tissue Oxygen Consumption and Learning/Memory Performance after Lateral Fluid Percussion Injury in Rats

2. Cyclophilins and their possible role in the stress response

3. Synaptic Mitochondria Are More Susceptible to Ca2+Overload than Nonsynaptic Mitochondria

4. Postinjury Cyclosporin A Administration Limits Axonal Damage and Disconnection in Traumatic Brain Injury

5. Cyclosporine A for neuroprotection: establishing dosing guidelines for safe and effective use

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