Targeting FEN1 Suppresses the Proliferation of Chronic Myeloid Leukemia Cells Through Regulating Alternative End-Joining Pathways-Reference-Cited by-同舟云学术

Targeting FEN1 Suppresses the Proliferation of Chronic Myeloid Leukemia Cells Through Regulating Alternative End-Joining Pathways

Published:2021-08-01 Issue:8 Volume:40 Page:1101-1111
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Zhu Yalin¹,Dai Hongdan¹,Wang Yonghong¹,Liang Yang¹,Feng Wenli¹,Yuan Ying²^ORCID

Affiliation:

1. Department of Laboratory Medicine, Key Laboratory of Laboratory Medical Diagnostics Designated by the Ministry of Education, Chongqing Medical University, Chongqing, China.

2. Department of Respiratory and Critical Care Medicine, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

https://www.liebertpub.com/doi/pdf/10.1089/dna.2021.0239

Reference45 articles.

1. Flap Endonuclease 1

2. Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase

3. Non-homologous DNA end joining and alternative pathways to double-strand break repair

4. p21Cip1/Waf1 disrupts the recruitment of human Fen1 by proliferating-cell nuclear antigen into the DNA replication complex.

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