Coculturing Human Islets with Proangiogenic Support Cells to Improve Islet Revascularization at the Subcutaneous Transplantation Site-Reference-Cited by-同舟云学术

Coculturing Human Islets with Proangiogenic Support Cells to Improve Islet Revascularization at the Subcutaneous Transplantation Site

Published:2016-02 Issue:3-4 Volume:22 Page:375-385
ISSN:1937-3341
Container-title:Tissue Engineering Part A
language:en
Short-container-title:Tissue Engineering Part A

Author:

Buitinga Mijke¹,Janeczek Portalska Karolina²,Cornelissen Dirk-Jan¹,Plass Jacqueline¹,Hanegraaf Maaike³,Carlotti Françoise³,de Koning Eelco³⁴⁵,Engelse Marten³,van Blitterswijk Clemens⁶,Karperien Marcel¹,van Apeldoorn Aart¹,de Boer Jan⁷

Affiliation:

1. Department of Developmental BioEngineering, MIRA Institute for Biomedical Technology and Technical Medicine, University of Twente, Enschede, The Netherlands.

2. Department of Tissue Regeneration, MIRA Institute for Biomedical Technology and Technical Medicine, University of Twente, Enschede, The Netherlands.

3. Department of Nephrology, Leiden University Medical Center, Leiden, The Netherlands.

4. Department of Endocrinology, Leiden University Medical Center, Leiden, The Netherlands.

5. Hubrecht Institute–Royal Netherlands Academy of Arts and Sciences (KNAW) and University Medical Center Utrecht, Utrecht, The Netherlands.

6. Department of Complex Tissue Regeneration, Institute for Technology Inspired Regenerative Medicine (MERLN), Maastricht University, Maastricht, The Netherlands.

7. Laboratory for Cell Biology-Inspired Tissue Engineering, Institute for Technology Inspired Regenerative Medicine (MERLN), Maastricht, The Netherlands.

Publisher

Mary Ann Liebert Inc

Subject

Biomedical Engineering,Biomaterials,Biochemistry,Bioengineering

Link

https://www.liebertpub.com/doi/pdf/10.1089/ten.tea.2015.0317

Reference55 articles.

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4. Elevated portal vein drug levels of sirolimus and tacrolimus in islet transplant recipients: local immunosuppression or islet toxicity?1

5. Production of tissue factor by pancreatic islet cells as a trigger of detrimental thrombotic reactions in clinical islet transplantation

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