Mechanisms and Consequences of Neuronal Stretch InjuryIn VitroDiffer with the Model of Trauma
Author:
Affiliation:
1. Department of Bioengineering. University of Pennsylvania, Philadelphia, Pennsylvania.
2. Department of Neurosurgery, University of Pennsylvania, Philadelphia, Pennsylvania.
Publisher
Mary Ann Liebert Inc
Subject
Clinical Neurology
Link
http://www.liebertpub.com/doi/pdf/10.1089/neu.2006.23.193
Reference40 articles.
1. NMDA Receptor Activation Contributes to a Portion of the Decreased Mitochondrial Membrane Potential and Elevated Intracellular Free Calcium in Strain-Injured Neurons
2. Enhanced Vulnerability to NMDA Toxicity in Sublethal Traumatic Neuronal Injury In Vitro
3. Strain measurements in cultured vascular smooth muscle cells subjected to mechanical deformation
4. The number of molecules taken up by electroporated cells: quantitative determination
5. Acute Alterations in [Ca2+]iin NG108-15 Cells Subjected to High Strain Rate Deformation and Chemical Hypoxia: Anin VitroModel for Neural Trauma
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