A Critical Analysis of the Role of the Neurotrophic Protein S100B in Acute Brain Injury
Author:
Affiliation:
1. Department of Neurosurgery, Georg August University, Göttingen, Germany.
2. Department of Neurosurgery, Virginia Commonwealth University Medical Center, Richmond, Virginia.
Publisher
Mary Ann Liebert Inc
Subject
Neurology (clinical)
Link
http://www.liebertpub.com/doi/pdf/10.1089/neu.2006.23.1185
Reference120 articles.
1. Relation between electroconvulsive therapy, cognitive side effects, neuron specific enolase, and protein S-100
2. S-100β protects cultured neurons against glutamate- and staurosporine-induced damage and is involved in the antiapoptotic action of the 5 HT1A-receptor agonist, Bay x 3702
3. NMDA Receptor Activation Contributes to a Portion of the Decreased Mitochondrial Membrane Potential and Elevated Intracellular Free Calcium in Strain-Injured Neurons
4. High Serum S100B Levels for Trauma Patients without Head Injuries
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