Angiotensin II-Induced Production of Mitochondrial Reactive Oxygen Species: Potential Mechanisms and Relevance for Cardiovascular Disease-Reference-Cited by-同舟云学术

Angiotensin II-Induced Production of Mitochondrial Reactive Oxygen Species: Potential Mechanisms and Relevance for Cardiovascular Disease

Published:2013-10 Issue:10 Volume:19 Page:1085-1094
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Dikalov Sergey I.¹,Nazarewicz Rafal R.¹

Affiliation:

1. Free Radicals in Medicine Core, Division of Clinical Pharmacology, Vanderbilt University Medical Center, Nashville, Tennessee.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2012.4604

Reference68 articles.

1. Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

2. Nox4 and Nox2 NADPH Oxidases Mediate Distinct Cellular Redox Signaling Responses to Agonist Stimulation

3. Disruption of the Ang II type 1 receptor promotes longevity in mice

4. Subcellular localization of Nox4 and regulation in diabetes

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