Molecular Mechanisms of the Crosstalk Between Mitochondria and NADPH Oxidase Through Reactive Oxygen Species—Studies in White Blood Cells and in Animal Models-Reference-Cited by-同舟云学术

Molecular Mechanisms of the Crosstalk Between Mitochondria and NADPH Oxidase Through Reactive Oxygen Species—Studies in White Blood Cells and in Animal Models

Published:2014-01-10 Issue:2 Volume:20 Page:247-266
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Kröller-Schön Swenja¹,Steven Sebastian¹,Kossmann Sabine¹²,Scholz Alexander¹,Daub Steffen¹,Oelze Matthias¹,Xia Ning³,Hausding Michael¹,Mikhed Yuliya¹,Zinßius Elena¹,Mader Michael¹,Stamm Paul¹,Treiber Nicolai⁴,Scharffetter-Kochanek Karin⁴,Li Huige³,Schulz Eberhard¹,Wenzel Philip¹²,Münzel Thomas¹,Daiber Andreas¹

Affiliation:

1. 2nd Medical Clinic, Department of Cardiology, Medical Center of the Johannes Gutenberg University, Mainz, Germany.

2. Center of Thrombosis and Hemostasis, Medical Center of the Johannes Gutenberg University, Mainz, Germany.

3. Department of Pharmacology, Medical Center of the Johannes Gutenberg University, Mainz, Germany.

4. Department of Dermatology and Allergic Diseases, University of Ulm, Ulm, Germany.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2012.4953

Reference69 articles.

1. Upregulation of Nox4 by Hypertrophic Stimuli Promotes Apoptosis and Mitochondrial Dysfunction in Cardiac Myocytes

2. Roles for Nox4 in the contractile response of bovine pulmonary arteries to hypoxia

3. Systemic administration of autologous, alloactivated helper-enriched lymphocytes to patients with metastatic melanoma of the lung

4. Properties of the Permeability Transition Pore in Mitochondria Devoid of Cyclophilin D

5. Subcellular localization of Nox4 and regulation in diabetes

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