Focal Adhesion Kinase: A Key Mediator of Transforming Growth Factor Beta Signaling in Fibroblasts
Author:
Affiliation:
1. Departments of Dentistry and Physiology and Pharmacology, University of Western Ontario, London, Canada.
Publisher
Mary Ann Liebert Inc
Subject
Critical Care and Intensive Care Medicine,Emergency Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/wound.2012.0363
Reference15 articles.
1. The complex dialogue between (myo)fibroblasts and the extracellular matrix during skin repair processes and ageing
2. Inhibition of focal adhesion kinase prevents experimental lung fibrosis and myofibroblast formation
3. Potential Therapeutic Targets for Cardiac Fibrosis
4. Myofibroblast Differentiation by Transforming Growth Factor-ॆ1 Is Dependent on Cell Adhesion and Integrin Signaling via Focal Adhesion Kinase
5. FAK Is Required for TGFβ-induced JNK Phosphorylation in Fibroblasts: Implications for Acquisition of a Matrix-remodeling Phenotype
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