V3 Loop of Human Immunodeficiency Virus Type 1 Suppresses Interleukin 2-Induced T Cell Growth
Author:
Publisher
Mary Ann Liebert Inc
Subject
Infectious Diseases,Virology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/aid.1997.13.151
Reference56 articles.
1. Isolation of a T-Lymphotropic Retrovirus from a Patient at Risk for Acquired Immune Deficiency Syndrome (AIDS)
2. The Human Immunodeficiency Virus: Infectivity and Mechanisms of Pathogenesis
3. Frequent Detection and Isolation of Cytopathic Retroviruses (HTLV-III) from Patients with AIDS and at Risk for AIDS
4. Increased viral burden and cytopathicity correlate temporally with CD4+ T-lymphocyte decline and clinical progression in human immunodeficiency virus type 1-infected individuals
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1. Selectively Impaired CD8+ but Not CD4+ T Cell Cycle Arrest during Priming as a Consequence of Dendritic Cell Interaction with Plasmodium-Infected Red Cells;The Journal of Immunology;2005-09-07
2. Activation of gp120 of human immunodeficiency virus by their V3 loop-derived peptides;Biochemical and Biophysical Research Communications;2002-09
3. Synthesis of an N-glucoasparagine analog as a building block for a V3-loop glycopeptide from GP120 of HIV-I;Carbohydrate Research;1998-12
4. T-Tropic Human Immunodeficiency Virus Type 1 (HIV-1)-Derived V3 Loop Peptides Directly Bind to CXCR-4 and Inhibit T-Tropic HIV-1 Infection;Journal of Virology;1998-12
5. V3 Loop of Human Immunodeficiency Virus Type 1 Reduces Cyclin E Expression and Induces G1Arrest in Interleukin 2-Dependent T Cells;AIDS Research and Human Retroviruses;1998-01
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