Differential Regulation of T Cell Receptor-Mediated Th1 Cell IFN-γProduction and Proliferation by Divergent cAMP-Mediated Redox Pathways
Author:
Affiliation:
1. Department of Pathology, The University of Connecticut Health Center, Farmington, CT 06030-3105
2. Department of Medicine, The University of Connecticut Health Center, Farmington, CT 06030-3105
Publisher
Mary Ann Liebert Inc
Subject
Virology,Cell Biology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/107999001753238033
Reference42 articles.
1. Cyclic AMP-dependent protein kinase type I mediates the inhibitory effects of 3‘,5‘-cyclic adenosine monophosphate on cell replication in human T lymphocytes.
2. Locus of inhibitory action of cAMP-dependent protein kinase in the antigen receptor-triggered cytotoxic T lymphocyte activation pathway.
3. Increased intracellular cyclic adenosine monophosphate inhibits T lymphocyte-mediated cytolysis by two distinct mechanisms.
4. Control of human T-lymphocyte interleukin-2 production by a cAMP-dependent pathway
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