HIV-1 VprB and C RNA Cleavage by Potent 10–23 DNAzymes That Also Cause Reversal of G2 Cell Cycle Arrest Mediated by Vpr Genes
Author:
Affiliation:
1. National Institute of Immunology, New Delhi-110067, India.
Publisher
Mary Ann Liebert Inc
Subject
Genetics,Molecular Biology,Molecular Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/oli.2007.0091
Reference17 articles.
1. Production of acquired immunodeficiency syndrome-associated retrovirus in human and nonhuman cells transfected with an infectious molecular clone
2. Inhibition of HIV-1 gene expression by novel DNA enzymes targeted to cleave HIV-1 TAR RNA: potential effectiveness against all HIV-1 isolates
3. Progression to AIDS in the absence of a gene for vpr or vpx
4. HIV-1 Vpr increases viral expression by manipulation of the cell cycle: A mechanism for selection of Vpr in vivo
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4. RNA-Cleaving DNA Enzymes and Their Potential Therapeutic Applications as Antibacterial and Antiviral Agents;From Nucleic Acids Sequences to Molecular Medicine;2012
5. Genetic and functional characterization of human immunodeficiency virus type 1 VprC variants from north India: presence of unique recombinants with mosaic genomes from B, C and D subtypes within the open reading frame of Vpr;Journal of General Virology;2009-11-01
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