Combined NOX/ROS/PKC Signaling Pathway and Metabolomic Analysis Reveals the Mechanism of TRAM34-Induced Endothelial Progenitor Cell Senescence-Reference-Cited by-同舟云学术

Combined NOX/ROS/PKC Signaling Pathway and Metabolomic Analysis Reveals the Mechanism of TRAM34-Induced Endothelial Progenitor Cell Senescence

Published:2021-06-07 Issue: Volume: Page:
ISSN:1547-3287
Container-title:Stem Cells and Development
language:en
Short-container-title:Stem Cells and Development

Author:

Cui Xiaodong¹²,Li Xiaoxia¹,He Yanting²,Yu Jie²,Fu Jie²,Song Bo³,Zhao Robert Chunhua¹^ORCID

Affiliation:

1. Department of Basic Medicine, Institute of Stem Cell and Regenerative Medicine, Qingdao University Medical College, Qingdao University, Qingdao, P.R. China.

2. School of Basic Medical Sciences, Weifang Medical University, Weifang, P.R. China.

3. School of Pharmacy, Weifang Medical University, Weifang, P.R. China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Developmental Biology,Hematology

Link

https://www.liebertpub.com/doi/pdf/10.1089/scd.2021.0062

Reference40 articles.

1. KCa3.1 inhibition switches the phenotype of glioma-infiltrating microglia/macrophages

2. Inhibition of vascular calcification by block of intermediate conductance calcium-activated potassium channels with TRAM-34

3. Macrophages facilitate post myocardial infarction arrhythmias: roles of gap junction and KCa3.1

4. The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans

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