Activation of CB2 Receptors by (−)-Cannabichromene but Not (+)-Cannabichromene-Reference-Cited by-同舟云学术

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Activation of CB2 Receptors by (−)-Cannabichromene but Not (+)-Cannabichromene

Published:2024-08-13 Issue: Volume: Page:
ISSN:2578-5125
Container-title:Cannabis and Cannabinoid Research
language:en
Short-container-title:Cannabis and Cannabinoid Research

Author:

Udoh Michael¹,Santiago Marina¹^ORCID,Haneef Syed¹,Rodger Alison²³,Marlowe Charles K.⁴,Barr Philip J.⁴,Connor Mark¹^ORCID

Affiliation:

1. Macquarie Medical School, Macquarie University, North Ryde, Australia.

2. School of Natural Sciences, Macquarie University, North Ryde, Australia.

3. Present address: Research School of Chemistry, Australian National University, ACT 2601, Australia.

4. A Division of InMed Pharmaceuticals, BayMedica, LLC, South San Francisco, California, USA.

Publisher

Mary Ann Liebert Inc

Link

https://www.liebertpub.com/doi/pdf/10.1089/can.2023.0212

Reference24 articles.

1. Inhibitory effect of cannabichromene, a major non-psychotropic cannabinoid extracted from Cannabis sativa, on inflammation-induced hypermotility in mice

2. Cannabichromene is a cannabinoid CB 2 receptor agonist

3. Plant-Derived Cannabinoids Modulate the Activity of Transient Receptor Potential Channels of Ankyrin Type-1 and Melastatin Type-8

4. Cannabichromene, Related Phytocannabinoids, and 5-Fluoro-cannabichromene Have Anticonvulsant Properties in a Mouse Model of Dravet Syndrome

5. The cannabinoid TRPA1 agonist cannabichromene inhibits nitric oxide production in macrophages and ameliorates murine colitis

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