Immune Control of γ-Herpesviruses
Author:
Affiliation:
1. School of Chemistry and Molecular Biosciences, University of Queensland and Brisbane, Australia.
2. Child Health Research Center, Brisbane, Australia.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Molecular Medicine,Immunology
Link
https://www.liebertpub.com/doi/pdf/10.1089/vim.2019.0080
Reference111 articles.
1. Varicella-Zoster Virus Infection of Human Dendritic Cells and Transmission to T Cells: Implications for Virus Dissemination in the Host
2. Vaccination with inactivated murine gammaherpesvirus 68 strongly limits viral replication and latency and protects type I IFN receptor knockout mice from a lethal infection
3. Epstein-Barr Virus–Infected Resting Memory B Cells, Not Proliferating Lymphoblasts, Accumulate in the Peripheral Blood of Immunosuppressed Patients
4. IFNγand Perforin Cooperate to Control Infection and Prevent Fatal Pathology During Persistent Gammaherpesvirus Infection in Mice
5. Postexposure vaccination massively increases the prevalence of gamma -herpesvirus-specific CD8+ T cells but confers minimal survival advantage on CD4-deficient mice
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1. A replication-deficient gammaherpesvirus vaccine protects mice from lytic disease and reduces latency establishment;npj Vaccines;2024-06-24
2. Indirect CD4 + T cell protection against mouse gamma-herpesvirus infection via interferon gamma;Journal of Virology;2024-05-14
3. Expression of the monocarboxylate transporter MCT1 is required for virus-specific mouse CD8 + T cell memory development;Proceedings of the National Academy of Sciences;2024-03-18
4. Replication-dead gammaherpesvirus vaccine protects against acute replication, reactivation from latency, and lethal challenge in mice;2023-09-26
5. Conquering the Host: Determinants of Pathogenesis Learned from Murine Gammaherpesvirus 68;Annual Review of Virology;2021-09-29
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