Impairments of Synaptic Plasticity in Aged Animals and in Animal Models of Alzheimer's Disease-Reference-Cited by-同舟云学术

Impairments of Synaptic Plasticity in Aged Animals and in Animal Models of Alzheimer's Disease

Published:2012-04 Issue:2 Volume:15 Page:235-238
ISSN:1549-1684
Container-title:Rejuvenation Research
language:en
Short-container-title:Rejuvenation Research

Author:

Balietti Marta¹²,Tamagnini Francesco³,Fattoretti Patrizia¹²,Burattini Costanza³,Casoli Tiziana¹,Platano Daniela⁴,Lattanzio Fabrizia⁵,Aicardi Giorgio³⁶

Affiliation:

1. Neurobiology of Aging Laboratory, INRCA Scientific Technological Area, Ancona, Italy.

2. Cellular Bioenergetics Laboratory, INRCA Scientific Technological Area, Ancona, Italy.

3. Department of Human and General Physiology, University of Bologna, Bologna, Italy.

4. Laboratory of Immunorheumatology and tissue regeneration, Istituto Ortopedico Rizzoli, Bologna, Italy.

5. Scientific Direction, INRCA Scientific Technological Area, Ancona, Italy.

6. Interdepartmental Centre “Luigi Galvani” for the Study of Biophysics, Bioinformatics and Biocomplexity, University of Bologna, Bologna, Italy.

Publisher

Mary Ann Liebert Inc

Subject

Geriatrics and Gerontology,Aging

Link

https://www.liebertpub.com/doi/pdf/10.1089/rej.2012.1318

Reference29 articles.

1. Long-Term Potentiation at CA3–CA1 Hippocampal Synapses with Special Emphasis on Aging, Disease, and Stress

2. Calcineurin Links Ca2+Dysregulation with Brain Aging

3. Cysteine regulation of protein function – as exemplified by NMDA-receptor modulation

4. Intracellular Redox State Alters NMDA Receptor Response during Aging through Ca2+/Calmodulin-Dependent Protein Kinase II

5. Reversal of Age-Related Alterations in Synaptic Plasticity by Blockade of L-Type Ca2+Channels

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