Vpr in Plasma of HIV Type 1-Positive Patients Is Correlated with The HIV Type 1 RNA Titers-Reference-Cited by-同舟云学术

Vpr in Plasma of HIV Type 1-Positive Patients Is Correlated with The HIV Type 1 RNA Titers

Published:2007-03 Issue:3 Volume:23 Page:391-397
ISSN:0889-2229
Container-title:AIDS Research and Human Retroviruses
language:en
Short-container-title:AIDS Research and Human Retroviruses

Author:

Hoshino Shigeki¹²,Sun Binlian¹,Konishi Mitsuru³,Shimura Mari¹,Segawa Tatsuya⁴,Hagiwara Yoshiaki⁴,Koyanagi Yoshio⁵,Iwamoto Aikichi⁶,Mimaya Jun-Ichi⁷,Terunuma Hiroshi⁸,Kano Shigeyuki¹²,Ishizaka Yukihito¹

Affiliation:

1. Research Institute, International Medical Center of Japan, Shinjuku-ku, Tokyo 162-8655, Japan.

2. Graduate School of Comprehensive Human Sciences, University of Tsukuba, Tsukuba 305-8577, Japan.

3. Center for Infectious Diseases, Nara Medical University, Kashihara, Nara 634-8522, Japan.

4. Immuno-Biological Laboratories, Co., Fujioka, Gunma 375-0005, Japan.

5. Laboratory of Viral Pathogenesis, Institute for Virus Research, Kyoto University, Sakyou-ku, Kyoto 606-8507, Japan.

6. Department of Infectious Diseases, The Institute of Medical Science, The University of Tokyo, Minato-ku 108-8639, Tokyo, Japan.

7. Department of Hematology and Oncology, Children's Hospital of Shizuoka Prefecture, Aoi-ku, Shizuoka 420-8660, Japan.

8. Biotherapy Institute of Japan, Koutou-ku, Tokyo, 135-0051, Japan.

Publisher

Mary Ann Liebert Inc

Subject

Infectious Diseases,Virology,Immunology

Link

http://www.liebertpub.com/doi/pdf/10.1089/aid.2006.0124

Reference18 articles.

1. Declining Morbidity and Mortality among Patients with Advanced Human Immunodeficiency Virus Infection

2. Identification of a Reservoir for HIV-1 in Patients on Highly Active Antiretroviral Therapy

3. Cytokine-Induced Expression of HIV-1 in a Chronically Infected Promonocyte Cell Line

4. Cytokines Alter Production of HIV-1 from Primary Mononuclear Phagocytes

5. The Vpr protein of human immunodeficiency virus type 1 influences nuclear localization of viral nucleic acids in nondividing host cells.

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