NFKB1−94 Insertion/Deletion ATTG Polymorphism Contributes to Risk of Systemic Lupus Erythematosus-Reference-Cited by-同舟云学术

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NFKB1−94 Insertion/Deletion ATTG Polymorphism Contributes to Risk of Systemic Lupus Erythematosus

Published:2012-04 Issue:4 Volume:31 Page:611-615
ISSN:1044-5498
Container-title:DNA and Cell Biology
language:en
Short-container-title:DNA and Cell Biology

Author:

Gao Ming¹,Wang Chun-Hui²,Sima Xiutian³,Han Xue-Mei⁴

Affiliation:

1. Department of Anesthesiology, China-Japan Union Hospital of Jilin University, Changchun, Jilin, People's Republic of China.

2. Department of Neurosurgery, Jilin Province Hospital, Changchun, Jilin, People's Republic of China.

3. Department of Neurosurgery, West China Hospital of Sichuan University, Chengdu, Sichuan, People's Republic of China.

4. Department of Neurology, China-Japan Union Hospital of Jilin University, Changchun, Jilin, People's Republic of China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Genetics,Molecular Biology,General Medicine

Link

http://www.liebertpub.com/doi/pdf/10.1089/dna.2011.1389

Reference38 articles.

1. The 65-kDa subunit of human NF-kappa B functions as a potent transcriptional activator and a target for v-Rel-mediated repression.

2. Nuclear Factor-κB — A Pivotal Transcription Factor in Chronic Inflammatory Diseases

3. A NFKB1 promoter polymorphism is involved in susceptibility to ulcerative colitis

4. Morbidity and Mortality in Systemic Lupus Erythematosus During a 10-Year Period

5. New Insights into the Role of Nuclear Factor-κB, a Ubiquitous Transcription Factor in the Initiation of Diseases

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