Prolactin-Deficient GH3B3 Cells Are Defective in the Utilization of the Endogenous Prolactin Promoter Yet Are Fully Competent to Initiate Transcription from a Transfected Prolactin Promoter
Author:
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Genetics,Molecular Biology,General Medicine
Link
http://www.liebertpub.com/doi/pdf/10.1089/dna.1991.10.105
Reference40 articles.
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4. Reconstitution of cell-type-specific transcription of the rat prolactin gene in vitro.
5. A Method for Isolation of Intact, Translationally Active Ribonucleic Acid
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3. CpG methylation represses the activity of the rat prolactin promoter in rat GH3 pituitary cell lines;Molecular and Cellular Endocrinology;1995-02
4. Dexamethasone-Mediated Induction of MMTV-myf5 in DD3 Myoblasts Increases Endogenous myogenin Expression but Does Not Transactivate myf5;Experimental Cell Research;1994-06
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