Mycobacterium tuberculosisPE_PGRS17 Promotes the Death of Host Cell and Cytokines Secretion via Erk Kinase Accompanying with Enhanced Survival of RecombinantMycobacterium smegmatis-Reference-Cited by-同舟云学术

Mycobacterium tuberculosisPE_PGRS17 Promotes the Death of Host Cell and Cytokines Secretion via Erk Kinase Accompanying with Enhanced Survival of RecombinantMycobacterium smegmatis

Published:2013-08 Issue:8 Volume:33 Page:452-458
ISSN:1079-9907
Container-title:Journal of Interferon & Cytokine Research
language:en
Short-container-title:Journal of Interferon & Cytokine Research

Author:

Chen Tian¹,Zhao Quanju¹,Li Wu¹,Xie Jianping¹

Affiliation:

1. Institute of Modern Biopharmaceuticals, State Key Laboratory Breeding Base of Eco-Environment and Bio-Resource of the Three Gorges Area, School of Life Sciences, Southwest University, Chongqing, China.

Publisher

Mary Ann Liebert Inc

Subject

Virology,Cell Biology,Immunology

Link

http://www.liebertpub.com/doi/pdf/10.1089/jir.2012.0083

Reference31 articles.

1. PE_PGRS Antigens ofMycobacterium tuberculosisInduce Maturation and Activation of Human Dendritic Cells

2. Are the PE-PGRS proteins of Mycobacterium tuberculosis variable surface antigens?

3. Execution of Macrophage Apoptosis by PE_PGRS33 of Mycobacterium tuberculosis Is Mediated by Toll-like Receptor 2-dependent Release of Tumor Necrosis Factor-α

4. Evasion of innate immunity by Mycobacterium tuberculosis: is death an exit strategy?

5. Permissiveness of Human Monocytes and Monocyte-Derived Macrophages to Infection by Promastigotes of Leishmania (Viannia) panamensis

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