Protein Oxidation and Lipid Peroxidation in Brain of Subjects with Alzheimer's Disease: Insights into Mechanism of Neurodegeneration from Redox Proteomics-Reference-Cited by-同舟云学术

Protein Oxidation and Lipid Peroxidation in Brain of Subjects with Alzheimer's Disease: Insights into Mechanism of Neurodegeneration from Redox Proteomics

Published:2006-11 Issue:11-12 Volume:8 Page:2021-2037
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Sultana Rukhsana¹,Perluigi Marzia²³,Butterfield D. Allan⁴

Affiliation:

1. Department of Chemistry, and Sanders-Brown Center on Aging, University of Kentucky, Lexington, Kentucky, U.S.A.

2. Department of Chemistry, University of Kentucky, Lexington, Kentucky, U.S.A.

3. Department of Biochemical Sciences, University of Rome "La Sapienza," Rome, Italy.

4. Department of Chemistry, Sanders-Brown Center on Aging, and Center of Membrane Sciences, University of Kentucky, Lexington, Kentucky, U.S.A.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2006.8.2021

Reference226 articles.

1. β-Amyloid Stimulation of Inducible Nitric-oxide Synthase in Astrocytes Is Interleukin-1β- and Tumor Necrosis Factor-α (TNFα)-dependent, and Involves a TNFα Receptor-associated Factor- and NFκB-inducing Kinase-dependent Signaling Mechanism

2. Oxidative Modification of Creatine Kinase BB in Alzheimer’s Disease Brain

3. Enhancement of β-Amyloid Peptide Aβ(1-40)-Mediated Neurotoxicity by Glutamine Synthetase

4. Protein oxidation in the brain in Alzheimer's disease

5. Increased Protein Oxidation and Decreased Creatine Kinase BB Expression and Activity after Spinal Cord Contusion Injury

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